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									<identifier>oai:www.peertechzpublications.org:10.17352/2455-5495.000009</identifier>
									<datestamp>2016-05-18</datestamp>
									<setSpec>PTZ.ARDM:VOL2</setSpec>
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									<oai_dc:dc xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance" xmlns:oai_dc="http://www.openarchives.org/OAI/2.0/oai_dc/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:mml="http://www.w3.org/1998/Math/MathML" xsi:schemaLocation="http://www.openarchives.org/OAI/2.0/oai_dc/ http://www.openarchives.org/OAI/2.0/oai_dc.xsd">
										<dc:title>
										Intractable Hematuria Due to Advanced Cardiac Failure and Venous Stasis
										</dc:title><dc:creator>Bora Özveren</dc:creator><dc:creator>Selçuk Yücel</dc:creator><dc:description>&lt;p&gt;Intractable&amp;nbsp;&amp;nbsp; gross&amp;nbsp;&amp;nbsp; hematuria&amp;nbsp;&amp;nbsp; without&amp;nbsp;&amp;nbsp; an&amp;nbsp;&amp;nbsp; obvious&amp;nbsp;&amp;nbsp; or&amp;nbsp;&amp;nbsp; common urologic&amp;nbsp;&amp;nbsp; pathology&amp;nbsp;&amp;nbsp; needs&amp;nbsp;&amp;nbsp; more&amp;nbsp;&amp;nbsp; effort&amp;nbsp;&amp;nbsp; for&amp;nbsp;&amp;nbsp; correct&amp;nbsp;&amp;nbsp; diagnosis&amp;nbsp;&amp;nbsp; and appropriate&amp;nbsp; management.&amp;nbsp; In&amp;nbsp; this&amp;nbsp; case,&amp;nbsp; we&amp;nbsp; report&amp;nbsp; on&amp;nbsp; a&amp;nbsp; patient&amp;nbsp; with intractable&amp;nbsp; hematuria&amp;nbsp; originating&amp;nbsp; from&amp;nbsp; varicose&amp;nbsp; vessels&amp;nbsp; of&amp;nbsp; urinary bladder due to advanced venous stasis after severe cardiac failure.&lt;/p&gt;&lt;p&gt;&lt;strong&gt;Case Report&lt;/strong&gt;&lt;/p&gt;&lt;p&gt;A 66-year-old male was consulted for massive hematuria and clot retention&amp;nbsp; subsequent&amp;nbsp; to&amp;nbsp; urethral&amp;nbsp; catheterization&amp;nbsp; after&amp;nbsp; admission&amp;nbsp; into intensive&amp;nbsp; care&amp;nbsp; unit&amp;nbsp; because&amp;nbsp; of&amp;nbsp; cardiac&amp;nbsp; decompensation.&amp;nbsp; His&amp;nbsp; medical history revealed cardiomyopathy and aortic metallic valve replacement 20&amp;nbsp; years&amp;nbsp; ago&amp;nbsp; and&amp;nbsp; gout&amp;nbsp; disease&amp;nbsp; for&amp;nbsp; 25&amp;nbsp; years.&amp;nbsp; He&amp;nbsp; had&amp;nbsp; been&amp;nbsp; on&amp;nbsp; oral anticoagulation&amp;nbsp; with&amp;nbsp; warfarin&amp;nbsp; for&amp;nbsp; 20&amp;nbsp; years.&amp;nbsp; On&amp;nbsp; physical&amp;nbsp; examination severe&amp;nbsp; ascites&amp;nbsp; and&amp;nbsp; pulmonary&amp;nbsp; effusion,&amp;nbsp; and&amp;nbsp; extensive&amp;nbsp; venous&amp;nbsp; stasis in&amp;nbsp; lower&amp;nbsp; trunk&amp;nbsp; was&amp;nbsp; found.&amp;nbsp; Central&amp;nbsp; venous&amp;nbsp; pressure&amp;nbsp; was&amp;nbsp; 30&amp;nbsp; cmH2O. His&amp;nbsp; blood&amp;nbsp; biochemistry&amp;nbsp; work-up&amp;nbsp; revealed&amp;nbsp; moderate&amp;nbsp; hyponatremia (132&amp;nbsp; meq/L)&amp;nbsp; and&amp;nbsp; a&amp;nbsp; serum&amp;nbsp; creatinin&amp;nbsp; of&amp;nbsp; 2.6&amp;nbsp; mg/dl&amp;nbsp; (normal&amp;nbsp; 0.5&amp;nbsp; to&amp;nbsp; 1.4). Anticoagulation was terminated at first. Prothrombin time / INR level returned&amp;nbsp; within&amp;nbsp; normal&amp;nbsp; limits&amp;nbsp; following&amp;nbsp; cessation&amp;nbsp; of&amp;nbsp; warfarin&amp;nbsp; and&amp;nbsp; he had only mild anemia. &lt;/p&gt;</dc:description>
										<dc:publisher>Archives of Renal Diseases and Management - Peertechz Publications</dc:publisher>
										<dc:date>2016-05-18</dc:date>
										<dc:type>Case Report</dc:type>
										<dc:identifier>https://doi.org/10.17352/2455-5495.000009</dc:identifier>
										<dc:language>en</dc:language>
										<dc:rights>Copyright © Bora Özveren et al.</dc:rights>
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